First of all, after I reread my original post, I mistakenly put the word glucose in the sentence about breaking down fat stores instead of the word energy. OOPSS Geez, sorry I committed such a crime. Second, Wheels dude, this is SUPPOSE to be a friendly forum...don't let it upset you so much. It's not really worth it.Originally posted by stumpy
Using the gluconeogenisis pathway, you body can break down fat stores and use them for glucose. This is not always a good idea though. One reason is because it takes a lot of ATP (a different kind of energy) to break the fat stores down.
Third:
http://muscle.ucsd.edu/musintro/fattyacid.shtml
Energy From Fatty Acids
Fat molecules consist of three fatty acid chains connected by a glycerol backbone. Fatty acids are basically long chains of carbon and hydrogen and are the major source of energy during normal activities.
Fatty acids are broken down by progressively cleaving two carbon bits and converting these to acetyl coenzyme A. The acetyl CoA is the oxidized by the same citric acid cycle involved in the metabolism of glucose. For every two carbons in a fatty acid, oxidation yields 5 ATPs generating the acetyl CoA and 12 more ATPs oxidizing the coenzyme. This makes fat a terrific molecule in which to store energy, as the body well knows (much to our dismay).
The only biological drawback to this, and other, forms of oxidative metabolism is its dependence on oxygen. Thus, if energy is required more rapidly than oxygen can be delivered, muscles switch to the less efficient anaerobic pathways. Interestingly, this implies that an anaerobic workout will not "burn" any fat, but will preferentially deplete the body of glucose. Of course, your body can't survive very long on just anaerobic metabolism...it just can't generate enough energy.
http://www.tri-ecoach.com/art3.htm
Most nutritional counselors recommend that sedentary individuals consume no more than 15% of their total calories as fat. Many exercise physiologists differ on this opinion, recommending anywhere from 15% up to 30%. The higher values may be necessary for endurance athletes given the amount of energy we need to fuel training and racing in addition to our everyday activities. During aerobic endurance training we burn some proportion of fat simultaneously with glycogen (the stored form of carbohydrates). The ratio of fat to carbohydrate we use is entirely speed dependent. At moderate speeds we utilize a higher proportions of fat to glycogen. As we go faster, the mix becomes progressively more biased toward glycogen usage.
This makes sense when we consider the example of the IronMan distance athlete's metabolism. Physiologists know that well-trained athletes can store only about 2000 (glycogen) calories within their muscle tissue and liver. This is enough to get you through about 2.5 hours of very intense racing. Athletes can't digest carbohydrates fast enough to keep up with the caloric demands of a high quality IronMan effort on glycogen stores alone. My relatively lean (~7% fat) body has approximately 32,000 calories of stored energy as fat (I can't safely use all the calories, but I won't need to). If I keep a reasonable pace, I have several times the caloric energy (with the help of stored fat) that I need to complete an IronMan. If I'm daring (or foolish) and go very (too) fast, I'll burn too high a proportion of carbs and only a little fat. This is the scenario where athletes bonk and "lose it" before the finish. Meanwhile, if I cruise along at a relatively comfortable pace, I can cover 140.6 miles in a day!
Peroxisome-proliferator-activated receptor delta activates fat metabolism to prevent obesity.
Wang YX, Lee CH, Tiep S, Yu RT, Ham J, Kang H, Evans RM
Gene Expression Laboratory, The Salk Institute, 10010 North Torrey Pines Road, La Jolla, CA 92037, USA.
In contrast to the well-established roles of PPARgamma and PPARalpha in lipid metabolism, little is known for PPARdelta in this process. We show here that targeted activation of PPARdelta in adipose tissue specifically induces expression of genes required for fatty acid oxidation and energy dissipation, which in turn leads to improved lipid profiles and reduced adiposity.